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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Endothelial nitric oxide synthase: a determinant of TNFalpha production by human monocytes/macrophages.

Until recently endothelial nitric oxide synthase (eNOS) has been associated exclusively with physiological functions, particularly in the cardiovascular system. However, increasing evidence has been accumulated that supports the concept for a role of eNOS in pathophysiology. In particular, detection of eNOS protein and activity in human monocytes/macrophages suggest an immunomodulatory role of this enzyme. Here, we review data that promote the hypothesis that by enhancing TNFalpha production, eNOS activity should be regarded as a novel pro-inflammatory parameter in human monocytes/macrophages.[1]

References

  1. Endothelial nitric oxide synthase: a determinant of TNFalpha production by human monocytes/macrophages. Mühl, H., Pfeilschifter, J. Biochem. Biophys. Res. Commun. (2003) [Pubmed]
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