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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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 Kaldis,  
 

Another Piece of the p27(Kip1) Puzzle.

How extracellular signals communicate with the cell cycle is poorly understood. In this issue, two papers () address this problem by reporting phosphorylation of the cyclin-dependent kinase inhibitor p27(Kip1) on a tyrosine residue by nonreceptor tyrosine kinases, which decreases p27 stability. This new mechanism could explain how cells enter the cell cycle from a quiescent state.[1]

References

  1. Another Piece of the p27(Kip1) Puzzle. Kaldis, P. Cell (2007) [Pubmed]
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