The world's first wiki where authorship really matters (Nature Genetics, 2008). Due credit and reputation for authors. Imagine a global collaborative knowledge base for original thoughts. Search thousands of articles and collaborate with scientists around the globe.

wikigene or wiki gene protein drug chemical gene disease author authorship tracking collaborative publishing evolutionary knowledge reputation system wiki2.0 global collaboration genes proteins drugs chemicals diseases compound
Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)



Gene Review

Egln2  -  egl-9 family hypoxia-inducible factor 2

Mus musculus

Synonyms: 0610011A13Rik, C85656, Egl nine homolog 2, Falkor, HIF-PH1, ...
Welcome! If you are familiar with the subject of this article, you can contribute to this open access knowledge base by deleting incorrect information, restructuring or completely rewriting any text. Read more.

Disease relevance of Egln2

  • The C-terminal domain of Falkor, isolated from the GSE library, has significant homology to a known human and rat cell growth regulator, SM-20, and to the C. elegans protein EGL-9, recently shown to modify the Hypoxia Inducible Factor-1alpha [1].

High impact information on Egln2

  • In this screen, expression of the C-terminal domain of Falkor conferred cells with resistance to cisplatin-induced growth arrest [1].

Biological context of Egln2


  1. Falkor, a novel cell growth regulator isolated by a functional genetic screen. Erez, N., Milyavsky, M., Goldfinger, N., Peles, E., Gudkov, A.V., Rotter, V. Oncogene (2002) [Pubmed]
WikiGenes - Universities