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UACA  -  uveal autoantigen with coiled-coil domains...

Bos taurus

 
 
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  • Motility was assessed by digital imaging microscopy before Western blot analysis, coimmunoprecipitation, or colocalization studies using ARF6, beta-actin, or beta-actin-binding protein-specific antibodies [1].
  • These results indicate that isoform-specific actin-binding proteins can be identified from cortical cytoplasm, and suggest that beta cap73 may not only act to spatially regulate the intracellular distribution of isoactins, but may also facilitate forward protrusion formation through the regulated release of free filament ends during cell motility [2].

References

  1. Betacap73-ARF6 interactions modulate cell shape and motility after injury in vitro. Riley, K.N., Maldonado, A.E., Tellier, P., D'Souza-Schorey, C., Herman, I.M. Mol. Biol. Cell (2003) [Pubmed]
  2. Beta cap73: a novel beta actin-specific binding protein. Shuster, C.B., Lin, A.Y., Nayak, R., Herman, I.M. Cell Motil. Cytoskeleton (1996) [Pubmed]
 
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