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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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ATP5G1  -  ATP synthase, H+ transporting,...

Homo sapiens

Synonyms: ATP synthase F(0) complex subunit C1, mitochondrial, ATP synthase lipid-binding protein, ATP synthase proteolipid P1, ATP synthase proton-transporting mitochondrial F(0) complex subunit C1, ATP5A, ...
 
 
 
 
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High impact information on ATP5G1

  • The observation that the mitochondrial ATPase subunit c protein accumulates in this disease suggests that autophagy, a pathway that regulates mitochondrial turnover, may be disrupted [1].
  • Real-time PCR demonstrated that all three nuclear gene isoforms (ATP5G1, ATP5G2, and ATP5G3) were consistently upregulated in the pancreas of alcohol-consuming rats, parallel with mitochondrial injury [2].

References

  1. Autophagy is disrupted in a knock-in mouse model of juvenile neuronal ceroid lipofuscinosis. Cao, Y., Espinola, J.A., Fossale, E., Massey, A.C., Cuervo, A.M., MacDonald, M.E., Cotman, S.L. J. Biol. Chem. (2006) [Pubmed]
  2. Rat mitochondrial ATP synthase ATP5G3: cloning and upregulation in pancreas after chronic ethanol feeding. Li, H.S., Zhang, J.Y., Thompson, B.S., Deng, X.Y., Ford, M.E., Wood, P.G., Stolz, D.B., Eagon, P.K., Whitcomb, D.C. Physiol. Genomics (2001) [Pubmed]
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