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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
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Disease relevance of Chlamydiaceae

  • The lesions induced in embryonated chicken eggs, the tinctoral properties, the ultrastructural morphology, the resistance of the organism to sodium sulfadiazine, and the presence of a chlamydial complement fixing antigen, identify this isolate as a member of the family Chlamydiaceae and suggest the agent to be Chlamydia psittaci [1].

High impact information on Chlamydiaceae

  • Our findings indicate that only some members of the family Chlamydiaceae have an arginine-responsive mechanism of gene regulation that is predicted to control arginine uptake from the host cell [2].
  • Trees for all five coding genes [the major outer-membrane protein (MOMP), GroEL chaperonin, KDO-transferase, small cysteine-rich lipoprotein and 60 kDa cysteine-rich protein] supported the current organization of the family Chlamydiaceae, which is based on ribosomal, biochemical, serological, ecological and DNA-DNA hybridization data [3].
  • The swabs and genital tracts were screened for Chlamydiae by a new 16S rRNA PCR and the sera by an ELISA for Chlamydiaceae lipopolysaccharide [4].


  1. Chlamydia psittaci induced pneumonia in a horse. McChesney, S.L., England, J.J., McChesney, A.E. The Cornell veterinarian. (1982) [Pubmed]
  2. Arginine-dependent gene regulation via the ArgR repressor is species specific in chlamydia. Schaumburg, C.S., Tan, M. J. Bacteriol. (2006) [Pubmed]
  3. Molecular evolution of the Chlamydiaceae. Bush, R.M., Everett, K.D. Int. J. Syst. Evol. Microbiol. (2001) [Pubmed]
  4. Diagnostic investigation into the role of Chlamydiae in cases of increased rates of return to oestrus in pigs. Camenisch, U., Lu, Z.H., Vaughan, L., Corboz, L., Zimmermann, D.R., Wittenbrink, M.M., Pospischil, A., Sydler, T. Vet. Rec. (2004) [Pubmed]
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