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MeSH Review: 

Cerebrovascular Trauma

 
 
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High impact information on Cerebrovascular Trauma

  • Most clinical and animal studies have shown alcohol to have a deleterious impact in the setting of cerebrovascular trauma; however, there are also data showing neuroprotective effects in low ethanol doses [1].
  • The findings of the present study suggest that VEGF, which appears to be increased in brain tissue during cerebrovascular trauma, increases the permeability of the BBB via the synthesis/release of nitric oxide and subsequent activation of soluble guanylate cyclase [2].
  • In the patients with traumatic or vascular brain injury, significant inverse correlations were present between the degree of neurological dysfunction, as indicated by the concomitant Glasgow coma score, and free NE, E, and DA and total NE and DA levels [3].

References

  1. Cerebral blood flow and dynamic cerebral autoregulation during ethanol intoxication and hypercapnia. Blaha, M., Aaslid, R., Douville, C.M., Correra, R., Newell, D.W. Journal of clinical neuroscience : official journal of the Neurosurgical Society of Australasia. (2003) [Pubmed]
  2. VEGF increases permeability of the blood-brain barrier via a nitric oxide synthase/cGMP-dependent pathway. Mayhan, W.G. Am. J. Physiol. (1999) [Pubmed]
  3. Free and total catecholamines in critical illness. Woolf, P.D., Hamill, R.W., Lee, L.A., McDonald, J.V. Am. J. Physiol. (1988) [Pubmed]
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