Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

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MeSH Review:

Lung Injury

Schreiber, T. et al., Chang, C.C. et al., Carraway, M.S. et al.

Barbara Schmidt, Waheedullah Karzai, Konrad Schwarzkopf, Torsten Schreiber, Lars Hueter, Elke Gaser, Martha S. Carraway, Hagir B. Suliman, Corrine Kliment, Karen E. Welty-Wolf, Tim D. Oury, Claude A. Piantadosi,

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Chemical compound and disease context of Lung Injury

There was no difference in left lung injury between dobutamine- and saline-infused animals, but right lung injury was aggravated in dobutamine-infused animals, as indicated by increased lung edema, histologic lung injury, and cell counts in lavage [1].
This is the first study to apply 1-D ge LC/MS/MS and experimental studies to reveal the mechanisms involved in the pathogenesis of ufCB-induced lung injury [2].
In all four lung injuries, mitochondrial fluorescence colocalized with mitochondrial superoxide dismutase, but not with proliferating cell nuclear antigen [3].

References

Effects of a catecholamine-induced increase in cardiac output on lung injury after experimental unilateral pulmonary acid instillation. Schreiber, T., Hueter, L., Gaser, E., Schmidt, B., Schwarzkopf, K., Karzai, W. Crit. Care Med. (2007) [Pubmed]
Proteomic analysis of proteins from bronchoalveolar lavage fluid reveals the action mechanism of ultrafine carbon black-induced lung injury in mice. Chang, C.C., Chen, S.H., Ho, S.H., Yang, C.Y., Wang, H.D., Tsai, M.L. Proteomics (2007) [Pubmed]
Mitochondrial biogenesis in the pulmonary vasculature during inhalational lung injury and fibrosis. Carraway, M.S., Suliman, H.B., Kliment, C., Welty-Wolf, K.E., Oury, T.D., Piantadosi, C.A. Antioxid. Redox Signal. (2008) [Pubmed]

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