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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Isoproterenol-induced myocardial injury resulting in altered S100A4 and S100A11 protein expression in the rat.

S-100 proteins (S100) are characterized by calcium-binding ability with two structural EF hands. Several S100 are expressed in cardiomyocytes and thought to play a crucial role in calcium signaling. To examine whether the expression of S100 is a response to detectable myocardial damage or regeneration, we investigated, immunohistochemically, the expression of S100A4 and S100A11 in the isoproterenol (ISP)-treated rat heart. Definite expression of S100A4 and S100A11 was demonstrated in normal cardiomyocytes, and their staining patterns were enhanced in the ISP-treated rat heart, suggesting the possible involvement of S1-A4 and S100A11 in ISP-induced myocardial damage.[1]

References

  1. Isoproterenol-induced myocardial injury resulting in altered S100A4 and S100A11 protein expression in the rat. Inamoto, S., Murao, S., Yokoyama, M., Kitazawa, S., Maeda, S. Pathol. Int. (2000) [Pubmed]
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