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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Deficiency of Mbd2 suppresses intestinal tumorigenesis.

Gene silencing through de novo methylation of CpG island promoters contributes to cancer. We find that Mbd2, which recruits co-repressor complexes to methylated DNA, is essential for efficient tumorigenesis in the mouse intestine. As Mbd2-deficient mice are viable and fertile, their resistance to intestinal cancer may be of therapeutic relevance.[1]

References

  1. Deficiency of Mbd2 suppresses intestinal tumorigenesis. Sansom, O.J., Berger, J., Bishop, S.M., Hendrich, B., Bird, A., Clarke, A.R. Nat. Genet. (2003) [Pubmed]
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