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Vaux, D.L. et al.

HtrA2/ Omi, a sheep in wolf's clothing.

Mammalian mitochondrial HtrA2/ Omi was originally described as an apoptosis inducer, but rather than having extra cells, mice with mutant HtrA2/ Omi suffer from a neurodegenerative disease due to progressive mitochondrial damage. This suggests that instead of promoting cell death by antagonizing inhibitor of apoptosis (IAP) proteins, the primary function of HtrA2/ Omi is to handle misfolded proteins in the mitochondria.[1]

References

HtrA2/Omi, a sheep in wolf's clothing. Vaux, D.L., Silke, J. Cell (2003) [Pubmed]

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