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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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A key stone cop regulates oxalate homeostasis.

A new study shows that Slc26a6-null mice manifest calcium-oxalate nephrolithiasis accompanied by enhanced net intestinal oxalate absorption. These findings point to a critical role for Slc26a6 in gastrointestinal oxalate secretion and suggest a genetic explanation for a common form of renal stone disease in humans.[1]

References

  1. A key stone cop regulates oxalate homeostasis. Kleta, R. Nat. Genet. (2006) [Pubmed]
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