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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Gene Review: 

CCKAR  -  cholecystokinin A receptor

Canis lupus familiaris

 
 
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High impact information on CCKAR

  • Moreover, pretreatment of the intraduodenal protein meal with the cholecystokinin-A receptor antagonist MK-329 abolished increases of S-28 and S-14 and caused a further twofold increase of gastric acid (P < 0.025) [1].
  • During the diluting period, inhibition of GB contractions by a CCKA receptor antagonist, atropine or hexamethonium, resulted in concentration of GB bile, whereas during the concentrating period, CCK-8 shifted the concentration process back to dilution [2].
  • In another study, intravenous administration of devazepide, a specific cholecystokinin-A receptor antagonist, at a dose of 0.1 mg/kg/hr was begun 15 min before postprandial saline intake and continued for 1 hr [3].

References

  1. Regulation of somatostatin-14 and -28 secretion by gastric acid in dogs: differential role of cholecystokinin. Greenberg, G.R., Fung, L., Pokol-Daniel, S. Gastroenterology (1993) [Pubmed]
  2. Relationship between gallbladder bile concentration and motility in conscious dogs: role of cholecystokinin. Muramatsu, S., Sonobe, K., Mizumoto, A., Yamada, T., Itoh, Z. Peptides (1997) [Pubmed]
  3. Postprandial normal saline intake delays gastric emptying of solids in conscious dogs: partial involvement of CCK in its mechanism. Tanaka, T., Mizumoto, A., Muramatsu, S., Mochiki, E., Haga, N., Suzuki, H., Itoh, Z. Dig. Dis. Sci. (1999) [Pubmed]
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