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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
 
 
 
 

HTLV-I-associated myelopathy: clinicopathologic correlation with localization of provirus to spinal cord.

A 49-year-old South African man developed a rapidly progressive myelopathy 14 months after blood transfusion and died 1 year after the onset of symptoms. Detailed pathologic examination of the spinal cord was consistent with the diagnosis of HTLV-I-associated myelopathy/tropical spastic paraparesis (HAM/ TSP). Although no HTLV-I viral particles, antigens, or nucleic acids were detected in situ, polymerase chain reaction assays revealed HTLV-I proviral DNA in cervical, thoracic, and lumbar levels of the spinal cord, with the greatest amount being detected at the thoracic level. These findings suggest that the pathogenesis of HAM/ TSP depends on direct infection of neural or immune elements within the spinal cord.[1]

References

  1. HTLV-I-associated myelopathy: clinicopathologic correlation with localization of provirus to spinal cord. Bhigjee, A.I., Wiley, C.A., Wachsman, W., Amenomori, T., Pirie, D., Bill, P.L., Windsor, I. Neurology (1991) [Pubmed]
 
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