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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Overproduction of transposon Tn10-encoded tetracycline resistance protein results in cell death and loss of membrane potential.

High-level expression of the Tn10 tetracycline resistance protein TetA in Escherichia coli caused partial collapse of the membrane potential, arrest of growth, and killing of the cells. Since alpha-methylglucoside transport was not affected, the overproduced TetA protein may cause not destruction of membrane structure but rather unrestricted translocation of protons and/or ions across the membrane.[1]

References

  1. Overproduction of transposon Tn10-encoded tetracycline resistance protein results in cell death and loss of membrane potential. Eckert, B., Beck, C.F. J. Bacteriol. (1989) [Pubmed]
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