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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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The influence of G-6-PD activity on the response of erythrocyte glutathione reductase to riboflavin deficiency.

Erythrocyte NAD(P)H2 glutathione oxidoreductase (EC 1.6.4.2.) activation coefficients (EGR AC) and D-glucose-6-phosphate: NADP 1-oxidoreductase, EC 1.1.1.49 (G-6-PD) activities were measured in 155 West African women to determine whether heterozygous G-6-PD deficiency produces false negative results in the EGR test for riboflavin deficiency. A positive correlation was found between EGR AC and G-6-PD activity demonstrating that heterozygous G-6-PD deficiency does result in abnormally depressed EGR AC values when G-6-PD activity is significantly reduced. However, heterozygous deficient genotypes with normal G-6-PD activity showed undepressed EGR AC values. These results indicate that, in its present form, the EGR test is not a suitable index of riboflavin status in subjects whose G-6-PD activity is less than 9 IU/g Hb when measured by the WHO-37 degrees assay.[1]

References

  1. The influence of G-6-PD activity on the response of erythrocyte glutathione reductase to riboflavin deficiency. Prentice, A.M., Bates, C.J., Prentice, A., Welch, S.G., Williams, K., McGregor, I.A. International journal for vitamin and nutrition research. Internationale Zeitschrift für Vitamin- und Ernährungsforschung. Journal international de vitaminologie et de nutrition. (1981) [Pubmed]
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