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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
 
 
 
 

Promotion by SR 48692 of gastric emptying and defaecation in rats suggesting a role of endogenous neurotensin.

We investigated the influence of the nonpeptide neurotensin receptor antagonist, SR 48692, administered orally, on gastric emptying and on acute defaecation. SR 48692 dose-dependently (ED50 approximately 0.7 microgram kg-1) increased gastric emptying of a food suspension, but it had no effect on that of a non-caloric meal. SR 48692 also dose-dependently promoted defaecation and increased faecal water content. We suggest that antagonism of endogenous neurotensin accounts for the gastric emptying and defaecation promoting action of SR 48692.[1]

References

  1. Promotion by SR 48692 of gastric emptying and defaecation in rats suggesting a role of endogenous neurotensin. Croci, T., Giudice, A., Manara, L., Gully, D., Le Fur, G. Br. J. Pharmacol. (1995) [Pubmed]
 
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