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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Platelet activation in patients with chronic pulmonary heart disease. Effect of dipyridamole treatment.

Plasma levels of beta-thromboglobulin (beta-TG), platelet factor 4(PF4) and platelet aggregation rate (PAR) were measured in remission phase of 15 patients affected with chronic pulmonary heart disease (CPHD). beta-TG, beta-TG/PF4, PF4 and PAR were significantly higher in the patients than in controls (P < 0.01 and 0.05, respectively). After 10 days of treatment with Dipyridamole 100mg tid, beta-TG, beta-TG/PF4 and PF4 decreased significantly compared with pretreatment values (P < 0.01 and 0.05, respectively). The results suggest that in vivo platelet activation is indeed present in patients with CPHD and that dipyridamole can antagonize platelet activation in vivo.[1]

References

  1. Platelet activation in patients with chronic pulmonary heart disease. Effect of dipyridamole treatment. Zhang, X., Ling, P. Chin. Med. J. (1995) [Pubmed]
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