The world's first wiki where authorship really matters (Nature Genetics, 2008). Due credit and reputation for authors. Imagine a global collaborative knowledge base for original thoughts. Search thousands of articles and collaborate with scientists around the globe.

wikigene or wiki gene protein drug chemical gene disease author authorship tracking collaborative publishing evolutionary knowledge reputation system wiki2.0 global collaboration genes proteins drugs chemicals diseases compound
Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Links

 

Gene Review

exp-2  -  Protein EXP-2

Caenorhabditis elegans

 
 
Welcome! If you are familiar with the subject of this article, you can contribute to this open access knowledge base by deleting incorrect information, restructuring or completely rewriting any text. Read more.
 

High impact information on exp-2

  • A gain-of-function mutation in the Caenorhabditis elegans exp-2 K(+)-channel gene is caused by a cysteine-to-tyrosine change (C480Y) in the sixth transmembrane segment of the channel (Davis, M.W., R. Fleischhauer, J.A. Dent, R.H. Joho, and L. Avery. 1999. Science. 286:2501-2504) [1].
  • Several loss-of-function mutations in exp-2 lead to broadening of the action potential and to a concomitant slowing of the pumping action of the pharynx [2].
 

Anatomical context of exp-2

References

  1. Dynamic interaction of S5 and S6 during voltage-controlled gating in a potassium channel. Espinosa, F., Fleischhauer, R., McMahon, A., Joho, R.H. J. Gen. Physiol. (2001) [Pubmed]
  2. Ultrafast inactivation causes inward rectification in a voltage-gated K(+) channel from Caenorhabditis elegans. Fleischhauer, R., Davis, M.W., Dzhura, I., Neely, A., Avery, L., Joho, R.H. J. Neurosci. (2000) [Pubmed]
 
WikiGenes - Universities