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Ohmori, E. et al.

Fluctuation of fetal rat hepatic histidine decarboxylase activity through the glucocorticoid-ACTH system.

Our studies suggest that the fluctuation of HDC activity in fetal liver in late gestation is regulated by the plasma glucocorticoid level through the pituitary-adrenal system. Taken together, these results support the conclusion that glucocorticoid promotes a rapid increase in HDC synthesis in fetal liver histamine-forming cells, as well as in mouse mastocytoma P-815 cells and rat glandular stomachs.[1]

References

Fluctuation of fetal rat hepatic histidine decarboxylase activity through the glucocorticoid-ACTH system. Ohmori, E., Imanishi, N., Ohgoh, M., Fukui, T., Ichikawa, A. Biochem. Pharmacol. (1991) [Pubmed]

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