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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

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Administration of GM1 ganglioside eliminates neuroleptic-induced sensorimotor deficits in MPTP-treated mice.

Injection of a low dose of haloperidol, that has no obvious behavioral effects in normal mice, produces akinesia, catalepsy, and sensory neglect in MPTP-treated mice. GM1 ganglioside treatment eliminates all of these behavioral impairments and also partially restores striatal dopamine content. These observations suggest that the MPTP-treated mouse may be a valuable model for studying mechanisms underlying parkinsonism and that administration of GM1 ganglioside may be an effective therapy.[1]

References

  1. Administration of GM1 ganglioside eliminates neuroleptic-induced sensorimotor deficits in MPTP-treated mice. Weihmuller, F.B., Hadjiconstantinou, M., Bruno, J.P., Neff, N.H. Neurosci. Lett. (1988) [Pubmed]
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