High impact information on Sentinel Turf Fungicide

• Laboratory strains of Mycosphaerella graminicola with decreased susceptibilities to the azole antifungal agent cyproconazole showed a multidrug resistance phenotype by exhibiting cross-resistance to an unrelated chemical, cycloheximide or rhodamine 6G, or both [1].
• Influence of pH on the stereoselective degradation of the fungicides epoxiconazole and cyproconazole in soils [2].
• Methanol extractives from the heartwood of all wood samples, except western hemlock, interfered with the quantitative determination of cyproconazole and tebuconazole using liquid chromatography (LC) with UV detection (LC-UV) [3].
• Chiral analysis suggested no significant selectivity in biotransformation for most of the compounds based on statistically unaltered enantiomer fractions (EFs) in the fish compared to food values; exceptions were a change in EF of myclobutanil and changes in diastereomer fractions (DFs) of propiconazole and cyproconazole [4].
• Increasing concentrations of the fungicides copper sulphate (CuSO(4)) and cyproconazole in the growth medium increased the proportion of ECMM in the biomass of Trametes versicolor and Gloeophyllum trabeum [5].

Associations of Sentinel Turf Fungicide with other chemical compounds

• The fungicide residues on grapes (cyproconazole, hexaconazole, kresoxim-methyl, myclobutanil, penconazole, tetraconazole, and triadimenol), the application rates of which were of a few tens of grams per hectare, were very low after treatment and were not detectable at harvest [6].
• Best results on in vitro fungal growth reduction were obtained with the four azoles tested, in particular with cyproconazole and hexaconazole, achieving 67-72% mycelial growth inhibition at the lowest dose [7].

Analytical, diagnostic and therapeutic context of Sentinel Turf Fungicide

• Progenies from a genetic cross between an isolate with an intermediate and a high sensitivity to azoles displayed a continuous range of phenotypes with respect to cyproconazole sensitivity, indicating that variation in azole sensitivity in this haploid organism is polygenic [8].

References