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Gene Review

PDCD7  -  programmed cell death 7

Homo sapiens

Synonyms: ES18, HES18, Programmed cell death protein 7, hES18
 
 
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Disease relevance of PDCD7

  • We further showed that transient overexpression of ES18 in mouse T-cell lymphoma increased the apoptotic cell death [1].
  • The expression of ES18 transcript was selectively regulated during the apoptosis of T-cell thymoma S49.1 induced by several stimuli [1].
  • Sensitivity or resistance to colicin M and phage ES18 and the electrophoretic pattern of the rRNA, which were incorporated into a recently proposed scheme for the identification of types of S. paratyphi B, individually or in combination fail to mark clones or other meaningful phylogenetic subdivisions [2].
 

High impact information on PDCD7

  • Characterization of a novel mouse cDNA, ES18, involved in apoptotic cell death of T-cells [1].
  • In addition, ES18 transcript was induced by C2-ceramide, which is a mediator of both dexamethasone- and staurosporine-induced apoptotic signaling [1].
  • One of the cDNAs, ES18, is preferentially expressed in lymph node and thymus, and contains noteworthy features of transcriptional regulator [1].
  • Interestingly, the ES18 transcript was differently regulated in the mutually antagonistic process, between dexamethasone- and A23187-induced cell death of T-cells [1].
  • Most d-tartrate-negative strains are members of an abundant, globally distributed clone (Pb 1) that is polymorphic for many biotype characters (including d-tartrate utilization), bacteriophage type, rRNA pattern, and colicin M and phage ES18 sensitivity [2].
 

Anatomical context of PDCD7

  • These data suggest that ES18 may be selectively involved in specific apoptotic processes in mouse T-cells [1].

References

  1. Characterization of a novel mouse cDNA, ES18, involved in apoptotic cell death of T-cells. Park, E.J., Kim, J.H., Seong, R.H., Kim, C.G., Park, S.D., Hong, S.H. Nucleic Acids Res. (1999) [Pubmed]
  2. Genetic population structure, clonal phylogeny, and pathogenicity of Salmonella paratyphi B. Selander, R.K., Beltran, P., Smith, N.H., Barker, R.M., Crichton, P.B., Old, D.C., Musser, J.M., Whittam, T.S. Infect. Immun. (1990) [Pubmed]
 
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