The world's first wiki where authorship really matters (Nature Genetics, 2008). Due credit and reputation for authors. Imagine a global collaborative knowledge base for original thoughts. Search thousands of articles and collaborate with scientists around the globe.

wikigene or wiki gene protein drug chemical gene disease author authorship tracking collaborative publishing evolutionary knowledge reputation system wiki2.0 global collaboration genes proteins drugs chemicals diseases compound
Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 

Editor

Share

Personal info

View

Links

About

Terms

 

 
 
 
 

In search of genes causing spontaneous hypertension.

1. Basal high blood pressure (BP) in the male and female F2 progeny produced by crosses between Wistar Kyoto rats (WKY/lzm) and stroke-prone spontaneously hypertensive rats (SHRSP/lzm) linked strongly with the leukosialin locus on chromosome 1. 2. Excess salt intake caused elevation of BP and the response was larger in the females than the males. 3. In the male F2 progeny, salt-sensitive hypertension significantly cosegregated with the RR1023 marker on chromosome 10, rather than with the angiotensin converting enzyme locus. 4. In the female F2 progeny, salt-sensitive hypertension significantly linked with the MitR244 marker on chromosome 3.[1]

References

  1. In search of genes causing spontaneous hypertension. Nara, Y., Ikeda, K., Matsumoto, C., Nabika, T., Sawamura, M., Yamori, Y. Clin. Exp. Pharmacol. Physiol. (1995) [Pubmed]
Annotations and hyperlinks in this abstract are from individual authors of WikiGenes or automatically generated by the WikiGenes Data Mining Engine. The abstract is from MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine.About WikiGenesOpen Access LicencePrivacy PolicyTerms of Useapsburg
 
WikiGenes - Universities