High impact information on OBP3

• This aberrant phenotype in red light requires functional phyB, suggesting that OBP3 is a positive regulator of phyB-mediated inhibition of hypocotyl elongation [1].
• The sob1-D mutant phenotype is caused by the overexpression of a Dof (DNA binding with one finger) transcription factor, OBF4 Binding Protein 3 (OBP3) [1].
• The Dof transcription factor OBP3 modulates phytochrome and cryptochrome signaling in Arabidopsis [1].
• Target genes for OBP3, a Dof transcription factor, include novel basic helix-loop-helix domain proteins inducible by salicylic acid [2].
• Two additional Dof proteins, called OBP2 and OBP3, were isolated through homologous screening [3].

Biological context of OBP3

• The severity of the growth defects correlated with OBP3 expression levels and in some cases led to death, suggesting that some Dof proteins play important roles in plant growth and development [3].

Associations of OBP3 with chemical compounds

• Characterization of salicylic acid-responsive, arabidopsis Dof domain proteins: overexpression of OBP3 leads to growth defects [3].

Analytical, diagnostic and therapeutic context of OBP3

• An alternate, though not mutually exclusive, model places OBP3 as a general inhibitor of tissue expansion with phyB and cry1, differentially modulating OBP3's role in this response [1].

References