The world's first wiki where authorship really matters (Nature Genetics, 2008). Due credit and reputation for authors. Imagine a global collaborative knowledge base for original thoughts. Search thousands of articles and collaborate with scientists around the globe.

wikigene or wiki gene protein drug chemical gene disease author authorship tracking collaborative publishing evolutionary knowledge reputation system wiki2.0 global collaboration genes proteins drugs chemicals diseases compound

Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

[search][options]

Editor

Personal info

View

About

Terms

Javascript disabled

Please enable Javascript support in your browser to use this application.

Instructions on how to enable JavaScript on different browsers can be found here: http://www.google.com/support/bin/answer.py?answer=23852.

[edit this page]

Gonzalez, M.J. et al.

Inorganic pentavalent arsenic methylation by rats: effect of concentration and dichromate.

The effect of the administered arsenic (As) dose and the dichromate ion (Cr) on the methylation process of arsenic was studied in rats exposed to arsenate to elucidate the biotransformation of As. After oral administration of different As (V) and Cr (VI) concentrations, the different As metabolites (inorganic, methylarsonic acid [MMA] and dimethylarsinic acid [DMA] were separated by cation-exchange chromatography and measured by atomic absorption spectrophotometry. Administration of high doses of As produced enzymatic saturation and non-enzymatic depletion, with decreases in DMA levels. The presence of the dichromate ion supported arsenate methylation as it favored reduction of As (V) to As (III), but Cr (VI) produced a significant decrease in the total As excreted.[1]

References

Inorganic pentavalent arsenic methylation by rats: effect of concentration and dichromate. Gonzalez, M.J., Aguilar, M.V., Martinez, M.C. Veterinary and human toxicology. (1995) [Pubmed]

Annotations and hyperlinks in this abstract are from individual authors of WikiGenes or automatically generated by the WikiGenes Data Mining Engine. The abstract is from MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine.About WikiGenes Open Access Licence Privacy Policy Terms of Use apsburg