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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
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Cidea-null mice had higher metabolic rate, lipolysis in BAT and core body temperature when subjected to cold treatment [1].
Conversely, TNF-alpha treatment decreased adipocyte CIDEA expression via the mitogen-activated protein kinase c-Jun NH(2)-terminal kinase [2].
Cell death-inducing DFFA (DNA fragmentation factor-alpha)-like effector A (CIDEA) belongs to a family of proapoptotic proteins that has five known members in humans and mice [2].
We investigated CIDEA in adipose tissue of obese and lean humans and mice [2].
We conclude that Cidea is a novel target gene for both PPARalpha and -gamma in the liver where these two transcription factors utilize the same PPRE region for dual regulation [3].
