Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)

Editor

Personal info

View

About

Terms

Javascript disabled

Please enable Javascript support in your browser to use this application.

Instructions on how to enable JavaScript on different browsers can be found here: http://www.google.com/support/bin/answer.py?answer=23852.

[edit this page]

Keltikangas-Jarvinen, L. et al.

Insulin resistance syndrome and autonomically mediated physiological responses to experimentally induced mental stress in adolescent boys.

We investigated the relationship between hemodynamic and other autonomically mediated responses to experimentally induced mental stress and the parameters of the insulin resistance syndrome (IRS) in 48 healthy adolescent boys. Mental stress was induced with mental arithmetic and the Stroop Color-Word Test. Heart rate (HR), finger blood volume (FBV), and skin conductance level (SCL) were recorded continuously during task performance. IRS parameters measured were serum insulin, high-density lipoprotein (HDL) cholesterol, serum triglyceride (TG), systolic blood pressure (SBP), subscapular skinfold (SSF), and subscapular to triceps skinfold ratio (STR). The results indicated that a high level and an increasing linear trend of HR and FBV during task performance were related, independently of each other and of body mass index (BMI), to a high insulin concentration. An increasing linear trend of HR during mental stress was also related to high SSFs independently of MI. In addition, a high SCL during task performance was associated with high TG levels, SSFs, and STRs. It is discussed whether stress-induced sympathetic overactivity might contribute to the development of the IRS.[1]

References

Insulin resistance syndrome and autonomically mediated physiological responses to experimentally induced mental stress in adolescent boys. Keltikangas-Jarvinen, L., Ravaja, N., Raikkonen, K., Lyytinen, H. Metab. Clin. Exp. (1996) [Pubmed]

Annotations and hyperlinks in this abstract are from individual authors of WikiGenes or automatically generated by the WikiGenes Data Mining Engine. The abstract is from MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine.About WikiGenes Open Access Licence Privacy Policy Terms of Use apsburg

The world's first wiki where authorship really matters (Nature Genetics, 2008). Due credit and reputation for authors. Imagine a global collaborative knowledge base for original thoughts. Search thousands of articles and collaborate with scientists around the globe.