High impact information on NSC 311

• Gene expression and microscopic analysis of Arabidopsis exposed to chloroacetanilide herbicides and explosive compounds. A phytoremediation approach [1].
• Dichloroacetamide safeners protect maize (Zea mays L.) against injury from chloroacetanilide and thiocarbamate herbicides [2].
• TaGSTF1, which resembled ZmGSTF1, the dominant GST in maize (Zea mays), was highly active in conjugating 1-chloro-2,4-dinitrobenezene (CDNB) but had low activities towards chloroacetanilide, diphenyl ether and aryloxphenoxypropionate herbicides [3].
• Enhanced tolerance of transgenic poplar plants overexpressing gamma-glutamylcysteine synthetase towards chloroacetanilide herbicides [4].
• Exposure of poplars to chloroacetanilide herbicides resulted in a marked induction of GST activity in upper leaf positions but not in middle and lower leaves [4].

Biological context of NSC 311

• The growth and the biomass of all poplar lines were markedly reduced by the two chloroacetanilide herbicides [4].
• Dialkylquinoneimine metabolites of chloroacetanilide herbicides induce sister chromatid exchanges in cultured human lymphocytes [5].
• Kinetics and mechanism of the nucleophilic displacement reactions of chloroacetanilide herbicides: investigation of alpha-substituent effects [6].

Anatomical context of NSC 311

• Glutathione-dependent cytotoxicity of the chloroacetanilide herbicides alachlor, metolachlor, and propachlor in rat and human hepatoma-derived cultured cells [7].

Gene context of NSC 311

• Analysis of the chloroacetanilide herbicides in water using SPME with CAR/PDMS and GC/ECD [8].

Analytical, diagnostic and therapeutic context of NSC 311

• Immunoassays were used as a toolto determine critical chloroacetanilide herbicides in water and soil samples without purification steps [9].
• Determination of chloroacetanilide herbicide metabolites in water using high-performance liquid chromatography-diode array detection and high-performance liquid chromatography/mass spectrometry [10].
• The limit of quantitation (LOQ) for all metabolites using the HPLC-DAD method was 0.20 microg/l, whereas the LOQ using the HPLC/MS method was at 0.05 microg/l. These metabolite-determination methods are valuable for acquiring information about water quality and the fate and transport of the parent chloroacetanilide herbicides in water [10].
• Rapid method for analysis of chloroacetanilide and chloronitrophenoxyaniline herbicides by gas chromatography [11].

References