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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
MeSH Review

Eschscholzia

 
 
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High impact information on Eschscholzia

  • The presented method allows detection and identification of isoquinoline alkaloids in crude methanolic extracts of medicinal plants as Eschscholzia californica CHAM [1].
  • Expression patterns of STM-like KNOX and Histone H4 genes in shoot development of the dissected-leaved basal eudicot plants Chelidonium majus and Eschscholzia californica (Papaveraceae) [2].
  • A genomic clone, bbe1 was isolated that encodes the methyl jasmonate-inducible berberine bridge enzyme of antimicrobial benzophenanthridine alkaloid biosynthesis in the California poppy Eschscholzia californica [3].
  • Four barbiturates, barbituric acid, butethal, phenobarbital, and 2-thiobarbituric acid, of fourteen tested were found to induce accumulation of benzophenanthridine alkaloids in cell suspension cultures of the California poppy Eschscholzia california [4].
  • This model, when adapted to crude extracts, permits the demonstration of both anti-lipoperoxidant and antihepatotoxic activity of reference products like quercetin and silymarin and plant extracts like Rosmarinus officinalis and Eschscholzia californica [5].
 

Associations of Eschscholzia with chemical compounds

  • A 70% ethanol extract of California poppy (Eschscholzia californica) was able to bind to 5-HT(1A) and 5-HT(7) receptors at 100 mug/mL [6].
  • Selective desensitization of jasmonate- and pH-dependent signaling in the induction of benzophenanthridine biosynthesis in cells of Eschscholzia californica [7].
  • An aqueous alcohol extract of Eschscholzia californica (Ec) has been evaluated for benzodiazepine, neuroleptic, antidepressant, antihistaminic and analgesic properties, in order to complete the study of the sedative and anxiolytic effects previously demonstrated [8].
 

Gene context of Eschscholzia

  • Alkaloids from Eschscholzia californica and their capacity to inhibit binding of [3H]8-Hydroxy-2-(di-N-propylamino)tetralin to 5-HT1A receptors in Vitro [6].

References

  1. Analysis of isoquinoline alkaloids in medicinal plants by capillary electrophoresis-mass spectrometry. Sturm, S., Stuppner, H. Electrophoresis (1998) [Pubmed]
  2. Expression patterns of STM-like KNOX and Histone H4 genes in shoot development of the dissected-leaved basal eudicot plants Chelidonium majus and Eschscholzia californica (Papaveraceae). Groot, E.P., Sinha, N., Gleissberg, S. Plant Mol. Biol. (2005) [Pubmed]
  3. Isolation and analysis of a gene bbe1 encoding the berberine bridge enzyme from the California poppy Eschscholzia californica. Hauschild, K., Pauli, H.H., Kutchan, T.M. Plant Mol. Biol. (1998) [Pubmed]
  4. Barbiturate induced benzophenanthridine alkaloid formation proceeds by gene transcript accumulation in the California poppy. Haider, G., Kislinger, T., Kutchan, T.M. Biochem. Biophys. Res. Commun. (1997) [Pubmed]
  5. tert-Butyl hydroperoxide-induced injury in isolated rat hepatocytes: a model for studying anti-hepatotoxic crude drugs. Joyeux, M., Rolland, A., Fleurentin, J., Mortier, F., Dorfman, P. Planta Med. (1990) [Pubmed]
  6. Alkaloids from Eschscholzia californica and their capacity to inhibit binding of [3H]8-Hydroxy-2-(di-N-propylamino)tetralin to 5-HT1A receptors in Vitro. Gafner, S., Dietz, B.M., McPhail, K.L., Scott, I.M., Glinski, J.A., Russell, F.E., McCollom, M.M., Budzinski, J.W., Foster, B.C., Bergeron, C., Rhyu, M.R., Bolton, J.L. J. Nat. Prod. (2006) [Pubmed]
  7. Selective desensitization of jasmonate- and pH-dependent signaling in the induction of benzophenanthridine biosynthesis in cells of Eschscholzia californica. Färber, K., Schumann, B., Miersch, O., Roos, W. Phytochemistry (2003) [Pubmed]
  8. Neurophysiological effects of an extract of Eschscholzia californica Cham. (Papaveraceae). Rolland, A., Fleurentin, J., Lanhers, M.C., Misslin, R., Mortier, F. Phytotherapy research : PTR. (2001) [Pubmed]
 
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