The world's first wiki where authorship really matters (Nature Genetics, 2008). Due credit and reputation for authors. Imagine a global collaborative knowledge base for original thoughts. Search thousands of articles and collaborate with scientists around the globe.

wikigene or wiki gene protein drug chemical gene disease author authorship tracking collaborative publishing evolutionary knowledge reputation system wiki2.0 global collaboration genes proteins drugs chemicals diseases compound
Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
MeSH Review

Duodenal Neoplasms

 
 
Welcome! If you are familiar with the subject of this article, you can contribute to this open access knowledge base by deleting incorrect information, restructuring or completely rewriting any text. Read more.
 

Disease relevance of Duodenal Neoplasms

  • BACKGROUND: Although only 5 per cent of patients with familial adenomatous polyposis (FAP) die from duodenal cancer, a recent study indicated that the mortality rate is much higher in patients with Spigelman stage IV disease [1].
  • Fifteen patients with pancreatic cancer (14 head, 1 body) and 1 patient with duodenal cancer, (with paraaortic adenopathy), were subjected to combination treatment with infusional 5-FU, bolus injection of mitomycin-C, and XRT (4 patients were treated off the protocol) [2].
 

High impact information on Duodenal Neoplasms

 

Biological context of Duodenal Neoplasms

 

Anatomical context of Duodenal Neoplasms

 

Analytical, diagnostic and therapeutic context of Duodenal Neoplasms

References

  1. Pylorus-preserving pancreaticoduodenectomy for advanced duodenal disease in familial adenomatous polyposis. Gallagher, M.C., Shankar, A., Groves, C.J., Russell, R.C., Phillips, R.K. The British journal of surgery. (2004) [Pubmed]
  2. Increased resectability of locally advanced pancreatic and periampullary carcinoma with neoadjuvant chemoradiotherapy. Weese, J.L., Nussbaum, M.L., Paul, A.R., Engstrom, P.F., Solin, L.J., Kowalyshyn, M.J., Hoffman, J.P. Int. J. Pancreatol. (1990) [Pubmed]
  3. Cell proliferation of the duodenal mucosa in patients affected by familial adenomatous polyposis. Santucci, R., Volpe, L., Zannoni, U., Paganelli, G.M., Poggi, B., Calabrese, C., Biasco, G. Gastroenterology (1997) [Pubmed]
  4. Human gastrin-releasing peptide receptor mediates sustained CREB phosphorylation and transactivation in HuTu 80 duodenal cancer cells. Qu, X., Xiao, D., Weber, H.C. FEBS Lett. (2002) [Pubmed]
  5. The human gastrin-releasing peptide receptor gene structure, its tissue expression and promoter. Xiao, D., Wang, J., Hampton, L.L., Weber, H.C. Gene (2001) [Pubmed]
  6. Liver tumour promotion by chemicals: models and mechanisms. Sarma, D.S., Rao, P.M., Rajalakshmi, S. Cancer Surv. (1986) [Pubmed]
  7. Duodenal cancer in patients with familial adenomatous polyposis (FAP): results of a 10 year prospective study. Groves, C.J., Saunders, B.P., Spigelman, A.D., Phillips, R.K. Gut (2002) [Pubmed]
  8. Duodenal cancer arising from the remaining duodenum after pylorus-preserving pancreatoduodenectomy for ampullary cancer in familial adenomatous polyposis. Murakami, Y., Uemura, K., Sasaki, M., Morifuji, M., Hayashidani, Y., Sudo, T., Sueda, T. J. Gastrointest. Surg. (2005) [Pubmed]
  9. Evidence for adenoma-carcinoma sequence in the duodenum of patients with familial adenomatous polyposis. The Leeds Castle Polyposis Group (Upper Gastrointestinal Committee). Spigelman, A.D., Talbot, I.C., Penna, C., Nugent, K.P., Phillips, R.K., Costello, C., DeCosse, J.J. J. Clin. Pathol. (1994) [Pubmed]
  10. Ability of FDG-PET to detect all cancers in patients with familial adenomatous polyposis, and impact on clinical management. van Kouwen, M.C., Drenth, J.P., van Krieken, J.H., van Goor, H., Friederich, P., Oyen, W.J., Nagengast, F.M. Eur. J. Nucl. Med. Mol. Imaging (2006) [Pubmed]
 
WikiGenes - Universities