Disease relevance of Isomethylsystox sulfoxide

• Histopathological and lipid changes in various organs of chicks (Gallus domesticus) caused by acute and chronic toxicity of metasytox-R and lebaycid [1].

High impact information on Isomethylsystox sulfoxide

• All the compounds except fensulfothion and oxydemeton-methyl induced cell-cycle delay in varying degrees [2].
• Significant increase of sister-chromatid exchanges (SCE) in V79 cells treated with 2 organophosphorus pesticides (OPP), fenthion and oxydemeton-methyl, was observed [2].
• Studies on the cytogenetic effects of oxydemetonmethyl in the human leukocyte and mouse micronucleus test systems [3].
• Teratogenesis associated with oxydemeton-methyl in the stage 12 chick embryo [4].
• Congenital anomalies associated with maternal exposure to oxydemeton-methyl [5].

Anatomical context of Isomethylsystox sulfoxide

• The study has established the central nervous system depressant effect and proconvulsant potential of oxydemeton-methyl which is widely used by the agricultural workers in the form of field spray [6].

Associations of Isomethylsystox sulfoxide with other chemical compounds

• The clearance values for hemoperfusion with coated activated charcoal were higher under the same trial conditions, the values being 83.70 ml/min for demeton-S-methyl sulfoxide and 87.84 ml/min for dimethoate [7].
• Whether or not extracorporeal hemodialysis or hemoperfusion with coated activated charcoal might be used in eliminating organophosphates following poisoning with nitrostigmine, demeton-S-methyl sulfoxide, or dimethoate was here examined [7].

Analytical, diagnostic and therapeutic context of Isomethylsystox sulfoxide

• The insecticide Metasystox-R was tested for mutagenic activity by a multiple bioassay [8].

References