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Gene Review: 

dpy-10  -  Protein DPY-10

Caenorhabditis elegans

 
 
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High impact information on dpy-10

  • Reduction-of-function mutations in two Caenorhabditis elegans genes, the receptor tyrosine kinase gene let-23 and the collagen gene dpy-10, both alter the AG at the end of a short (ca. 50-nucleotide) intron to AA [1].
  • The genes do not, however, appear to be functionally redundant, because a dpy-10 null mutant is not rescued by the dpy-2 gene [2].
  • The nature of this mutation, combined with genetic data, indicates that DLRol is the null phenotype of dpy-10 [2].
 

Anatomical context of dpy-10

References

  1. Splicing in Caenorhabditis elegans does not require an AG at the 3' splice acceptor site. Aroian, R.V., Levy, A.D., Koga, M., Ohshima, Y., Kramer, J.M., Sternberg, P.W. Mol. Cell. Biol. (1993) [Pubmed]
  2. Molecular and genetic analyses of the Caenorhabditis elegans dpy-2 and dpy-10 collagen genes: a variety of molecular alterations affect organismal morphology. Levy, A.D., Yang, J., Kramer, J.M. Mol. Biol. Cell (1993) [Pubmed]
  3. Identification of a Novel Gene Family Involved in Osmotic Stress Response in Caenorhabditis elegans. Wheeler, J.M., Thomas, J.H. Genetics (2006) [Pubmed]
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