Psychiatry related information on Cardiomyopathy, Alcoholic

• In the United States, in both sexes and all races, long-term heavy alcohol consumption (of any beverage type) is the leading cause of a nonischemic, dilated cardiomyopathy, herein referred to as alcoholic cardiomyopathy (ACM) [1].

High impact information on Cardiomyopathy, Alcoholic

• These data suggest that elevated cardiac ACA exposure due to enhanced ADH expression may play an important role in the development of alcoholic cardiomyopathy [2].
• Alcoholic cardiomyopathy is characterized by impaired ventricular function although its toxic mechanism is unclear [2].
• Alcoholic cardiomyopathy in rats: inotropic responses to phenylephrine, glucagon, ouabain, and dobutamine [3].
• Using an animal model of alcoholism, we investigated whether 4 months of ethanol consumption was associated with a preclinical stage of alcoholic cardiomyopathy (ACM), as well as activation of the renin-angiotensin system and natriuretic peptides (NPs) [4].
• The functional state of the creatine kinase system of myocardial mitochondria in alcoholic cardiomyopathy [5].

Chemical compound and disease context of Cardiomyopathy, Alcoholic

• Long-standing ethanol consumption acts as a chronic cardiac stress and often leads to alcoholic cardiomyopathy [6].
• Chronic alcoholic cardiomyopathy. Protection of the isolated ischaemic working heart by ribose [7].
• 2. In accordance with the data obtained by Kino (J. molec, cell. Cardiol. 13, 5-12, 1981), when ethanol (36% of dietary calories) and aminotriazole were simultaneously administered an alcoholic cardiomyopathy developed while in the liver moderate fatty degeneration was revealed [8].

Biological context of Cardiomyopathy, Alcoholic

• Changes in cardiac signal transduction systems in chronic ethanol treatment preceding the development of alcoholic cardiomyopathy [9].

References