Disease relevance of Sulfhemoglobinemia

• Acute and chronic exposures to hydroxylamine have caused methemoglobinemia and sulfhemoglobinemia [1].
• Because the cyanosis did not improve after the patient received oxygen and methylene blue, sulfhemoglobinemia was suspected and confirmed by spectrophotometer analysis [2].
• Hemolytic anemia and sulfhemoglobinemia due to phenacetin abuse: a case with multivisceral adverse effects [3].

High impact information on Sulfhemoglobinemia

• Phenazopyridine-induced sulfhemoglobinemia: inadvertent rechallenge [4].
• Metoclopramide-induced sulfhemoglobinemia [5].
• We studied the incidence of methemoglobinemia, sulfhemoglobinemia and cytochrome b5 reductase deficiency in nine consecutive patients enrolled in a phase II trials using Sulofenur [6].
• An adolescent case of sulfhemoglobinemia associated with high-dose metoclopramide and N-acetylcysteine [7].
• Delayed sulfhemoglobinemia after acute dapsone intoxication [8].

Chemical compound and disease context of Sulfhemoglobinemia

• Sulfhemoglobinemia is suspected when a cyanotic patient has normal to near-normal oxygen tension, laboratory reports of elevated methemoglobin, and does not respond to methylene blue therapy [2].
• Phenacetin-induced sulfhemoglobinemia: report of a case and review of the literature [9].
• Sulfhemoglobinemia after dermal application of DMSO [10].
• The origin of sulfhemoglobinemia in man is discussed and the possible role of intraerythrocytic glutathione is emphasized [3].
• The reviewed medical literature suggests that the cyanosis of chronic phenacetin or acetanilid use is generally due to sulfhemoglobinemia rather than methemoglobinemia [9].

Anatomical context of Sulfhemoglobinemia

• OBJECTIVE: To report a case of sulfhemoglobinemia in a patient receiving phenazopyridine for a urinary tract infection [2].

References