High impact information on Blind Loop Syndrome

• Glycoprotein degradation in the blind loop syndrome: identification of glycosidases in jejunal contents [1].
• Histology and glucose hydrogen breath test were suggestive of a bacterial overgrowth syndrome in the small bowel, but standard culture-based techniques and serology failed to identify the causative bacteria [2].
• Impaired intestinal sodium and chloride transport in the blind loop syndrome of the rat [3].
• Breath hydrogen production after oral lactose loading was examined in infants and children with stagnant loop syndrome, blind loop syndrome, or both [4].
• Malabsorption of vitamin B12 and fat due to a stagnant loop syndrome was found in four of seven patients examined for this [5].

Chemical compound and disease context of Blind Loop Syndrome

• Value of urinary simple phenol and indican determinations in the diagnosis of the stagnant loop syndrome [6].
• When applied as diagnostic tests for the stagnant loop syndrome, the phenol excretion showed 2 false negative results, the p-cresol excretion 3 false negative and 2 false positive results, and the indican excretion 6 false positive results [6].
• Five patients with blind loop syndrome (Billroth II) were examined by measuring 14CO2 specific activity of expired breath samples taken at intervals after a meal containing glycine-1-14C cholate [7].

Anatomical context of Blind Loop Syndrome

• Pathogenesis of mucosal injury in the blind loop syndrome. Brush border enzyme activity and glycoprotein degradation [8].

References