Disease relevance of Microfilming

• Probably, in relation to the complex pathomechanisms responsible for the generation of myelofibrosis, the changing content of reticulin fibres was usually accompanied by corresponding alterations in the number of CD61+ megakaryocytes, including atypical microforms and precursor cells [1].
• A microform cleft lip has three major components: (1) a minor defect of the upper vermilion border with loss of the mucocutaneous ridge; (2) a narrow ridge of tissue, resembling an exaggerated philtral column extending to the nostril sill; and (3) a deformity of the nostril [2].
• These midline anomalies are recognized minimal manifestations (microforms) of holoprosencephaly [3].
• Correlations between microforms of the Van der Woude syndrome and cleft palate [4].

High impact information on Microfilming

• We report here that mice homozygous for targeted mutations of Cdon display the hallmark facial defects associated with microforms of HPE [5].
• The same microform was also predominant in adult AT, whereas the more reactive microform in respect to Con A predominates in adult fetuin [6].
• A total of 311 CD61-positive megakaryocytes, including precursors and atypical microforms, were evaluated in pre-treatment specimens derived from 11 patients with Ph1+ CML [7].
• Microtia: a microform of hemifacial microsomia [8].
• Linear blood flow velocity in brain, muscle capillaries and hepatic sinusoids was measured by means of microfilming in normal and hypoxemic rats (breathing with 7% O2 in N2) [9].

Associations of Microfilming with chemical compounds

• The offshoots of the Library are discussed, including the International Microform Journal of Legal Medicine, which was the first journal of its type in medicine [10].

Gene context of Microfilming

• Thus the question arises: do the CE always represent microforms of the Van der Woude syndrome, or are they only pathogenic similarities of a variable etiology [4]?
• The significance of microforms of CLP for anomalies and malformations of the jaws and face [11].
• In hyperthyroid rats, a significant reduction (37% relative to controls) in the concanavalin A-non reactive microform of AFP, was observed [12].
• Correlation of hypodontia with systemic disease leads to the hypothesis that this frequent dental anomaly may in some cases be a microform of systemic ectodermal dysplasia [13].

References