Disease relevance of Adolescent Development

• PURPOSE: The changing expectations, therapies and outcomes for young people with cystic fibrosis (CF) necessitate a re-evaluation of the impact of this chronic illness on adolescent development [1].

Psychiatry related information on Adolescent Development

• Their adolescent development was assessed by clinical staging, plus determinations of serum FSH, LH, testosterone and estradiol during both wakefulness and sleep [2].
• The groups included Substance Abuse Group, which promoted the 12-step model; Health Group; Psychotherapy Group; Leisure Time Group; Self-Awareness Group; and Multiple Family Group. The effect of the multiple groups was to provide a variety of experiences focusing on varied aspects of normal and dysfunctional adolescent development [3].

High impact information on Adolescent Development

• Human aggrecan keratan sulfate undergoes structural changes during adolescent development [4].
• We conclude that during adolescent development, Ang II may be an important factor in the regulation of salt and water metabolism, but not renal hemodynamics [5].
• In conclusion, circulating estrogen levels are very low in all boys prepubertally and rise steadily during adolescent development [6].
• Adolescent development alters stressor-induced Fos immunoreactivity in rat brain [7].
• These results suggest that testosterone induction of aromatase is critical for the organization of mature SI behavior in male rats over adolescent development [8].

Chemical compound and disease context of Adolescent Development

• This study examined the influence of adolescent development and pubertal changes in gonadal function on the development of norepinephrine (NE) turnover in the hypothalamus and cerebral cortex of rats from late juvenile to young adult ages [9].

Gene context of Adolescent Development

• Our results provide support for tailoring preventive interventions to the unique needs of children with ADHD and their parents at various stages of adolescent development [10].
• Despite continued impaired weight growth, pubertal patients do attain essentially normal gonadotrophin secretory responses to LHRH administration and are similar to subjects with constitutional delayed adolescent development in reproductive endocrine physiology [11].
• Data from these studies show clearly that exposure to high doses of multiple AAS during adolescent development can predispose animals to intense bouts of aggressive behavior during young adulthood [12].
• During normal adolescent development in males testosterone induces the adolescent growth spurt, at least in part, by increasing GH production via an increase in the amplitude of the peaks of GH which are released, and not by increasing the frequency of GH pulses [13].
• Adolescent development: advice in ABC of adolescence is potentially misleading [14].

References