High impact information on Musculocutaneous Nerve

• Using nerve stimulation, the musculocutaneous nerve was identified first and blocked with 10 mL of 1.5% lidocaine with 1:400,000 epinephrine [1].
• In contrast to vehicle, BDNF treatment reduced the loss and atrophy of motor neurons and enhanced the regrowth axon sprouts into the distal stump of musculocutaneous nerve [2].
• Adding 50 microg clonidine to the median and musculocutaneous nerves resulted in a significant increase in the duration of sensory block in these nerves (P < 0.0001) [3].
• Anesthesia was achieved in 52%-86% of the evaluated six nerves in the ropivacaine group and in 36%-87% in the bupivacaine group; the lowest figures were seen in the musculocutaneous nerve [4].
• The action of CO2 on phrenic (inspiratory) and internal intercostal (expiratory) motoneurons was compared with that exerted on non-respiratory motoneurons of the musculocutaneous nerve [5].

Biological context of Musculocutaneous Nerve

• In Groups A and B, no significant impairment of the donor median nerve function was found in a grasping test and the muscle contraction force of the flexor carpi radialis muscle, and histologic evaluation of the musculocutaneous nerve showed multiple regenerated axons distal to the coaptation site [6].
• In this study, 13 patients with unilateral brachial plexus injuries who had previously undergone conventional electromyographic testing underwent somatosensory evoked potential recording with median, ulnar, radial, musculocutaneous nerve and C5, C6, C7, C8 and T1 dermatomal stimulation [7].

Anatomical context of Musculocutaneous Nerve

• Based on an anatomic study, a transfer of the brachialis muscle branch of the musculocutaneous nerve (BMBMCN) to finger flexor functional fascicles of the median nerve was designed [8].
• Histologic results showed that the IGF-II treated group had higher axon counts and greater myelin thickness in the reinnervated musculocutaneous nerve [9].

Associations of Musculocutaneous Nerve with chemical compounds

• The Clonidine group (n = 30) received 10 mL of plain mepivacaine 1.5% for each nerve, but the median and musculocutaneous nerves also received a dose of 50 microg clonidine [3].
• Thirteen phenol injections to the musculocutaneous nerve were performed for excessive elbow flexion [10].
• OBJECTIVE: To evaluate the effectiveness of alcohol in neurolysis of the musculocutaneous nerve for the treatment of elbow flexor spasticity in individuals with a stroke [11].
• There was a poor correlation, however, between the presence of absence of SNAPs in the musculocutaneous nerve and the location of the lesion to the C5 root [12].
• The flexors of the elbow are controlled by the musculocutaneous nerve formed mainly from C5 and C6 fibers, the motor cells of which are usually located above the level of the spinal lesion [13].

Analytical, diagnostic and therapeutic context of Musculocutaneous Nerve

• We investigated the effect of selective application of clonidine to the median and musculocutaneous nerves during midhumeral block, a technique allowing selective nerve blocks with the use of different local anesthetics [3].
• If necessary, a lateral subcutaneous injection of 2 mL of 1.5% mepivacaine was administered at the wrist crease in the musculocutaneous nerve area [14].
• At the early time point of 1 week following the musculocutaneous nerve to biceps muscle implantation, there was no increased production of recombinant BDNF at the distal implanted musculocutaneous nerve, as assessed by Western blot analysis [15].

References