Disease relevance of Bulbar Palsy, Progressive

• Fifty-five MND patients, clinically divided into 22 amyotrophic lateral sclerosis (ALS), 18 ALS with probable UMN signs (ALS-PUMNS), 10 pure lower motor neuron syndrome (LMNS), and 5 progressive bulbar palsy (PBP), underwent standard TMS, recording from abductor digiti minimi and flexor allucis muscles [1].
• A Japanese man developed bulbar palsy and generalized neurogenic muscular atrophy with symmetrical hyperreflexia at the age of 57 and died 4.5 years after the onset [2].
• The authors present a case of acute intermittent porphyria (AIP) in an almost fatal relapse with quadriplegia, bulbar paralysis and coma [3].
• The patient presented with carpal tunnel syndrome, cardiomyopathy, bulbar palsy, dysphonia and polyneuropathy [4].
• A case of acute pulmonary edema and bulbar paralysis after local epinephrine infiltration [5].

High impact information on Bulbar Palsy, Progressive

• He postulates that phenothiazine-induced bulbar palsy may cause laryngeal and/or pharyngeal dysfunctions that are responsible for aspiration [6].
• Delayed post-irradiation bulbar palsy in nasopharyngeal carcinoma [7].
• We examined the serum levels of various IgG antiganglioside antibodies in the sera of 16 patients with GBS with bulbar palsy [GBS-BP(+)] and 72 patients with GBS without bulbar palsy [GBS-BP(-)] [8].
• Young age at onset, sporadic occurrence, sensorineural deafness, bulbar palsy, diffuse atrophy with weakness of limbs and progressive but benign course were the striking features [9].
• Nearly all reports on nonfatal cases describe severe clinical syndromes with tetraparesis, bulbar palsy, and coma [10].

Chemical compound and disease context of Bulbar Palsy, Progressive

• Bulbar palsy aggravated by metrizamide CT cisternography [11].

Gene context of Bulbar Palsy, Progressive

• Progressive bulbar palsy was not found; that diagnosis usually denotes merely the bulbar onset of ALS [12].
• We report a 64-year-old woman with follicular thyroid cancer found by subacute bulbar palsy [13].
• Infantile botulism classically presents with a triad of clinical features: Bulbar palsies (slow/absent pupil response) Alert Absent fever Other common features are: Constipation, ptosis and poor feeding The diagnosis is a clinical one, confirmed by EMG and by testing stool for the organism, C. botulinum, or its toxin [14].

Analytical, diagnostic and therapeutic context of Bulbar Palsy, Progressive

• In a hospital series of 70 patients on follow-up after radiotherapy for nasopharyngeal carcinoma, 14 patients (20%) developed delayed post-irradiation bulbar palsy 1 to 18 years after radiotherapy (mean 5.5 years) [7].

References