The world's first wiki where authorship really matters (Nature Genetics, 2008). Due credit and reputation for authors. Imagine a global collaborative knowledge base for original thoughts. Search thousands of articles and collaborate with scientists around the globe.

wikigene or wiki gene protein drug chemical gene disease author authorship tracking collaborative publishing evolutionary knowledge reputation system wiki2.0 global collaboration genes proteins drugs chemicals diseases compound
Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
MeSH Review

Erythrocytes, Abnormal

Welcome! If you are familiar with the subject of this article, you can contribute to this open access knowledge base by deleting incorrect information, restructuring or completely rewriting any text. Read more.

Disease relevance of Erythrocytes, Abnormal


High impact information on Erythrocytes, Abnormal

  • In addition, the abnormal erythrocytes from patients with PNH had fewer periodate oxidizable constituents than did normal erythrocytes, indicating a relative deficiency of cell-surface sialic acid [6].
  • AMr 73,000 protein isolated from the normal erythrocyte membranes of one PNH patient by using anti-DAF IgG was largely absent from the abnormal erythrocytes of this individual, suggesting that PNH cells lack the DAF protein [7].
  • Intravascular red blood cell (RBC) destruction is caused by increased sensitivity of the abnormal erythrocyte to complement-mediated lysis, due to the GPI absence of a membrane-bound glycosylphosphatidylinositol (GPI)-linked protein, which functions as an inhibitor of reactive lysis (CD59) [8].
  • The casein and histone kinase activities of normal and abnormal erythrocytes declined significantly with increasing age and buoyant density in Stractan density gradients [9].
  • Resolution of iron deficiency was defined as the restoration of transferrin saturation, serum ferritin level, anemia, and abnormal erythrocyte indexes to normal or baseline values [10].

Biological context of Erythrocytes, Abnormal


Associations of Erythrocytes, Abnormal with chemical compounds


Gene context of Erythrocytes, Abnormal


  1. Structural and functional differences between decay-accelerating factor and red cell acetylcholinesterase. Sugarman, J., Devine, D.V., Rosse, W.F. Blood (1986) [Pubmed]
  2. Erythrocyte morphology in women with severe preeclampsia and eclampsia. Preliminary observations with scanning electron microscopy. Cunningham, F.G., Lowe, T., Guss, S., Mason, R. Am. J. Obstet. Gynecol. (1985) [Pubmed]
  3. Enteric protein loss measured by fecal alpha 1-antitrypsin clearance in the assessment of Crohn's disease activity: a study of children and adolescents. Griffiths, A.M., Drobnies, A., Soldin, S.J., Hamilton, J.R. J. Pediatr. Gastroenterol. Nutr. (1986) [Pubmed]
  4. Tocopherol isomers in intravenous lipid emulsions and resultant plasma concentrations. Gutcher, G.R., Lax, A.A., Farrell, P.M. JPEN. Journal of parenteral and enteral nutrition. (1984) [Pubmed]
  5. Release of prostacyclin after erythrocyte adhesion to cultured vascular endothelium. Wautier, J.L., Pintigny, D., Maclouf, J., Wautier, M.P., Corvazier, E., Caen, J. J. Lab. Clin. Med. (1986) [Pubmed]
  6. Abnormality of glycophorin-alpha on paroxysmal nocturnal hemoglobinuria erythrocytes. Parker, C.J., Soldato, C.M., Rosse, W.F. J. Clin. Invest. (1984) [Pubmed]
  7. Deficiency of an erythrocyte membrane protein with complement regulatory activity in paroxysmal nocturnal hemoglobinuria. Pangburn, M.K., Schreiber, R.D., Müller-Eberhard, H.J. Proc. Natl. Acad. Sci. U.S.A. (1983) [Pubmed]
  8. Correction of the PNH defect by GPI-anchored protein transfer. Sloand, E.M., Maciejewski, J.P., Dunn, D., Moss, J., Brewer, B., Kirby, M., Young, N.S. Blood (1998) [Pubmed]
  9. Protein kinases and membrane protein phosphorylation in normal and abnormal human erythrocytes: variation related to mean cell age. Fairbanks, G., Palek, J., Dino, J.E., Liu, P.A. Blood (1983) [Pubmed]
  10. Intravenous iron dextran therapy in patients with iron deficiency and normal renal function who failed to respond to or did not tolerate oral iron supplementation. Barton, J.C., Barton, E.H., Bertoli, L.F., Gothard, C.H., Sherrer, J.S. Am. J. Med. (2000) [Pubmed]
  11. Erythrocyte anion transporter and antibrain immunoreactivity in chorea-acanthocytosis. A contribution to etiology, genetics, and diagnosis. Bosman, G.J., Bartholomeus, I.G., De Grip, W.J., Horstink, M.W. Brain Res. Bull. (1994) [Pubmed]
  12. Removal of uraemic plasma factor(s) using different dialysis modalities reduces phosphatidylserine exposure in red blood cells. Bonomini, M., Ballone, E., Di Stante, S., Bucciarelli, T., Dottori, S., Arduini, A., Urbani, A., Sirolli, V. Nephrol. Dial. Transplant. (2004) [Pubmed]
  13. Abnormal erythrocyte metabolism in hepatic disease: effect of NADP repletion. Smith, J.R., Kay, N.E., Gottlieb, A.J., Oski, F.A. Am. J. Hematol. (1979) [Pubmed]
  14. Abnormal erythrocyte pyrimidine nucleotides in uremic subjects. Angle, C.R., Swanson, M.S., Stohs, S.J., Markin, R.S. Nephron (1985) [Pubmed]
  15. Laboratory and field comparisons of adenosine influx in Plasmodium falciparum and Plasmodium vivax infected erythrocytes with genetic abnormalities from patients in Myanmar. Myint-Oo, n.u.l.l., O'Sullivan, W.J., Gero, A.M. Southeast Asian J. Trop. Med. Public Health (1997) [Pubmed]
  16. The erythrocyte skeletons of beta-adducin deficient mice have altered levels of tropomyosin, tropomodulin and EcapZ. Porro, F., Costessi, L., Marro, M.L., Baralle, F.E., Muro, A.F. FEBS Lett. (2004) [Pubmed]
  17. Thiol group modulation of sodium-lithium countertransport kinetics in diabetic nephropathy. Jones, S.C., Thomas, T.H., Marshall, S.M. Diabetologia (1997) [Pubmed]
  18. Multiple acyl-CoA dehydrogenase deficiency occurring in pregnancy and caused by a defect in riboflavin metabolism in the mother. Study of a kindred with seven deaths in infancy: Value of riboflavin therapy in preventing this syndrome. Harpey, J.P., Charpentier, C., Goodman, S.I., Darbois, Y., Lefèbvre, G., Sebbah, J. J. Pediatr. (1983) [Pubmed]
  19. Abnormal erythrocyte anion exchange in Alzheimer disease. Greco, F.A., Satlin, A., Solomon, A.K. Arch. Pathol. Lab. Med. (2000) [Pubmed]
  20. Absence of abnormal erythrocyte superoxide dismutase, copper, or zinc levels in patients with retinitis pigmentosa. Crouch, R.K., Chambers, J.K. The British journal of ophthalmology. (1982) [Pubmed]
WikiGenes - Universities