Disease relevance of Ultraviolet Rays

• A second patient, who had not showered immediately after leaving the whirlpool, was left with areas of skin hyperpigmentation and scarring despite treatment with ultraviolet rays and tetracycline [1].

High impact information on Ultraviolet Rays

• Autonomous growth of human T-lymphotropic virus type I infected human lymphocytes treated with N-methyl-N'-nitro-N-nitrosoguanidine and ultraviolet rays [2].
• The formation of fatty acid esters of vitamin D3 was demonstrated in rat skin exposed to artificial ultraviolet rays by using multi-dimensional high-performance liquid chromatography, ultraviolet spectrophotometry, and gas-liquid chromatography-mass spectrometry [3].
• However, the biological role of Sfn in cellular response to various stressors such as UV-C (mainly 254 nm wavelength ultraviolet ray) remains unclear [4].
• Similarly, neither pre-incubation nor illumination by ultraviolet ray could balance the inhibitory effect of SNP [5].
• In cell culture, caffeine has been shown to enhance the lethality of DNA-damaging agents including ultraviolet rays, X-irradiation, and alkylating agents [6].

Associations of Ultraviolet Rays with chemical compounds

• This finding suggests not only that ultraviolet rays could not penetrate deeply into the in vivo skin, but that the restriction of cutaneous synthesis of vitamin D3 observed in vivo may arise from this reduced penetration of ultraviolet rays [3].
• Other abiotic elicitors, such as ultraviolet rays and heavy metals, can trigger stilbene production [7].
• Tryptophan in charred fish or animal meat, ultraviolet rays, and irradiation, which damage genes of normal cells, have long been regarded as inducers of carcinoma, and agents such as alcohol, tobacco, aflatoxin, and nitrosoamine as promoters, with tobacco having both activities [8].
• Phospholipid metabolism in inflamed tissue of the mouse skin which had been induced by the application of 1-chloro-2, 4-dinitrobenzene (DNCB), croton oil, or irradiation of ultraviolet rays was examined, and it was found that phospholipid levels had increased in theinflamed tissues [9].
• The combined treatment with beta-propiolactone and ultraviolet rays described for the sterilization of HBsAg-positive sera does not interfere with the purification procedures for the production of HBs antigen [10].

Gene context of Ultraviolet Rays

• The presence of pinguecula would be an index of local irradiation of ultraviolet rays and decreased antioxidant activities [11].
• The parameters studied were: a)the action of chemicals tested in scalar doses on DNA synthesis of rat thymocytes; b)damage exerted by pesticides on human lymphocyte DNA measured as unscheduled DNA sythesis; c)the interference of chemicals tested with human lymphocyte repair capability after damage exerted on cells by ultraviolet rays [12].
• Melanocortin receptor type 1 (MC-1R) is an important control point for ultraviolet ray (UVR)-induced tanning response in the skin [13].
• "Toast Them, Burn Them, Roast Them". Ultraviolet ray therapy: the panacea of the 1920s and 1930s [14].

Analytical, diagnostic and therapeutic context of Ultraviolet Rays

• After treatment of carotene for 12 weeks, animals were exposed to ultraviolet ray (UV), and squalene monohydroperoxide (SqOOH)/squalene (Sq) ratios in the skin lipid were analyzed using the chemiluminescence-HPLC method [15].

References