Disease relevance of ADG

• In the third growth phase ADG was correlated with muscle fiber number (r = .4149, P < .001) and was not correlated with birth weight [1].
• The effects of cyadox and olaquindox were assessed in terms of fecal shedding of E. coli, the number of intraepithelial lymphocytes (IEL), immunoglobulin A-positive cells (APC) in the intestinal lamina propria, and ADG [2].
• RESULTS: Average daily gain was higher for CW (121 g/d; p < .05) compared with SI piglets 85 g/d); SH pig weight gain was intermediate (109 g/d) [3].
• Average daily gain for pigs not infected was 40% greater (P less than .01) than that of pigs given 20,000 S. ransomi larvae/kg body weight [4].

High impact information on ADG

• Zinc oxide increased (P < 0.05) ADG, ADFI, and G:F from d 0 to 10, and ADG and ADFI from d 10 to 24 [5].
• These results suggest that pigs from 13 to 20 and from 20 to 30 kg of BW fed diets containing 14.5 MJ of ME/kg had maximum N retention and ADG at 0.85 and 0.77 g of apparent ileal digestible lysine/MJ of ME, respectively [6].
• Average daily gain, IMF percentages, and water-holding capacity (WHC) were also determined [7].
• Effects of direct heterosis significantly increased weights at birth, 14, 56, 70, and 154 d of age in Exp. 1. Effects of direct heterosis significantly increased ADG from birth to 14, 28 to 56, and 70 to 154 d of age in Exp. 1. In Exp. 2, effect of direct heterosis significantly increased weights and ADG at all ages [8].
• Average daily gain was calculated for intervals between weights, and ultrasonic backfat measurements (A-mode) were taken at 154 d of age [8].

Chemical compound and disease context of ADG

• Average daily gain and total weight gain did not differ between groups; however, feed efficiency differed (P < 0.05) between lipopolysaccharide and control groups [9].

Biological context of ADG

• Average daily gain increased similarly in the three genotypes (125 g/d) [10].

Associations of ADG with chemical compounds

• Increasing dietary lysine improved d 0 to 14 ADG (P < .01), but no differences were observed for the overall experiment [11].
• The results showed pigs treated with 10 mg/kg cadmium significantly decreased average daily gain (ADG) (p<0.05) and increased feed/gain ratio (F/G) (p<0.05) compared to the control [12].
• Average daily gain, ADFI, gain/feed, backfat thickness, stomach keratinization score, blood pCO2, plasma urea N, and digestibilities of DM and N were not affected by treatment (P < .15) [13].
• Average daily gain responded quadratically (P < .05) to levels of Cu histidinate; maximum growth was seen in the 5% group [14].
• Average daily gain, ADFI, gain/feed, and fasting (15 to 18 h) plasma glucose and total protein concentrations were not affected (P > .10) by dietary treatment [15].

Other interactions of ADG

• Selection for increased ADG is expected to improve WHC but to decrease the percentage of LM water, with an associated decrease in juiciness [16].
• Neither backfat nor ADG was correlated with loin depth (r(g) = 0.0), and their mutual genetic correlation was 0.27 [17].
• Average daily gain was most depressed in pigs given the highest dose of virulent PRV plus P multocida [18].
• Average daily gain after weaning for AA genotype on IGF-1 locus was 20.58 g (P = 0.0347) higher than for AB [19].

Analytical, diagnostic and therapeutic context of ADG

• Average daily gain of the CS group was greater (P < 0.05) than the control group during wk 3 [20].

References