Disease relevance of Maxillary Artery

• In 3 dogs with intractable epistaxis, embolization of the terminal portion of the maxillary artery was performed with polyvinyl alcohol particles and contrast slurry [1].
• Hypercapnia or injections of isoproterenol and acetylcholine into the internal maxillary arteries increased blood flow and decreased middle cerebral arterial pressure, whereas injections of norepinephrine decreased blood flow and increased postrete pressure [2].

High impact information on Maxillary Artery

• The normal cerebral vasoconstriction obtained with injections of norepinephrine directly into the internal maxillary artery was unaffected during the diazoxide induced-hypotension [3].
• Injections of diazoxide (1-27 mg) into the internal maxillary artery produced dose-dependent increases in cerebral blood flow, an increase of 90% occurring with the highest dose [3].
• Administration of phentolamine, propranolol, or atropine into the internal maxillary artery did not modify the cerebrovascular response to diazoxide [3].
• Infusion of 1 to 12 mU of vasopressin into the internal maxillary artery of unanesthetized goats caused dose-dependent reductions in cerebral blood flow, a decrease of 36 +/- 4.7% (mean +/- S.E.) occurring with the highest dose [4].
• Small doses of 5-HT (0.1-9 micrograms) injected directly into the internal maxillary artery produced dose-dependent reductions in cerebral blood flow, a decrease of 49.8 +/- 2.5% (S.E.M.) occurring with the highest dose [5].

Biological context of Maxillary Artery

• Continuous infusion of 8-phenyltheophylline (8-PT), 100 micrograms/min, into the internal maxillary artery did not alter the resting cerebral blood flow or the cerebrovascular resistance, but significantly inhibited the cerebral vasodilation induced by adenosine, AMP, ADP and, to a lesser degree, ATP [6].

Anatomical context of Maxillary Artery

• 1. Injection of CO2-saturated saline in a distal direction into either a vertebral artery or an internal maxillary artery in pentobarbitone-anaesthetized cats produced abrupt changes in respiration [7].
• The effect of noradrenaline (NA) on mucociliary activity in the rabbit maxillary sinus was investigated in vivo by injecting it at increasing dosages (10(-11) to 10(-4) mol/kg) into the maxillary artery, the mucociliary response being recorded photoelectrically [8].
• Microinfusion of L-dopamine into the third cerebral ventricle (c.v.) or into the internal maxillary artery in intact as well as in lesioned lactating ewes depressed distinctly the prolactin level in the p.bl [9].

Associations of Maxillary Artery with chemical compounds

• The injection of adenosine, AMP, ADP and ATP (3-30 micrograms) directly into the internal maxillary artery increased cerebral blood flow and decreased cerebrovascular resistance in a dose-dependent manner [6].
• 3. Phenyl diguanide injected into either a vertebral or an internal maxillary artery also produced abrupt effects on respiration [7].
• Electrical stimulation of cervical sympathetic nerve produced reductions in cerebral blood flow which were significantly diminished during continuous infusion of GABA (20-40 micrograms/min) into the internal maxillary artery [10].
• Low doses of histamine (0.5-5.0 nmol/min) resulted in an increase in blood flow through the ipsilateral internal maxillary artery (IMA), which could be mainly attributed to a significant elevation of the CAP flow [11].
• After administration of phentolamine into the internal maxillary artery, stimulation produced increase in cerebral blood flow [12].

Gene context of Maxillary Artery

• THP (5 mg/day) was infused into the lingual or maxillary artery one hour before radiation on days 1-5 and 8-12, while intravenous 5-FU (150 mg/m2/day) was instilled continuously on days 1-5, 8-12, 15-19, and 22-26 in accordance with the radiation schedule (2 Gy/day) [13].

References