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MeSH Review

Electric Stimulation

 
 
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Disease relevance of Electric Stimulation

 

Psychiatry related information on Electric Stimulation

 

High impact information on Electric Stimulation

  • Electrophysiological recordings from flight muscles in the giant fiber pathway of adult eas flies reveal that induction of paralysis with electrical stimulation results in a brief seizure, followed by a failure of the muscles to respond to giant fiber stimulation [11].
  • The relaxation caused by either electrical stimulation or nitric oxide was enhanced by a selective inhibitor of cyclic guanosine monophosphate (GMP) phosphodiesterase (M&B 22,948) [12].
  • Here we describe the repeated induction of this experience by focal electrical stimulation of the brain's right angular gyrus in a patient who was undergoing evaluation for epilepsy treatment [13].
  • In the present paper, we report that the relative importance of cholinergic and non-cholinergic transmission in the regulation of TH activity varies with the pattern of electrical stimulation of the preganglionic nerve trunk [14].
  • The results of the present studies support this hypothesis, as we find that a stable enkephalin analogue (D-Ala 2,D-Leu5-enkephalin) inhibits the calcium-dependent release of vasopressin evoked by electrical stimulation of the rat pituitary stalk in vitro [15].
 

Chemical compound and disease context of Electric Stimulation

 

Biological context of Electric Stimulation

 

Anatomical context of Electric Stimulation

 

Associations of Electric Stimulation with chemical compounds

 

Gene context of Electric Stimulation

  • 1. Endothelin-1 and endothelin-3 enhanced concentration-dependently the rat vas deferens twitch response to electrical stimulation, endothelin-1 being three times more potent [36].
  • Using a preparation of adult isolated dorsal horn with dorsal roots attached, we found that electrical stimulation of roots induced a concomitant release of BDNF and an increased phosphorylation of NR1, which was partly prevented by the BDNF sequestering molecule, TrkB-IgG [37].
  • In contrast, antidromic electrical stimulation of the saphenous nerve caused a substantially greater release of SP in the skin of NK1(-/-) mice than in NK1(+/+) mice (P<0.05, n=5 to 6 mice/strain) [38].
  • The pyramidal neurons recorded in Otx1-/- mice responded to near-threshold electrical stimulation of the underlying white matter, with aberrant polysynaptic excitatory potentials often leading to late action potential generation [39].
  • The contractile responses, which were mediated predominantly by activation of noradrenergic nerves following electrical stimulation, were inhibited by bovine pancreatic polypeptide (BPP), human pancreatic polypeptide (HPP), avian pancreatic polypeptide (APP) and NPY in a dose-dependent manner using a constant flow bath [40].
 

Analytical, diagnostic and therapeutic context of Electric Stimulation

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