Disease relevance of Hematoma, Subdural, Chronic

• The levels of platelet-activating factor (PAF) and lipid metabolites related to choline glycerophospholipid were measured in the plasma and hematoma samples obtained from patients with chronic subdural hematoma [1].
• A triad of mental changes unsteady gait and urinary incontinence, was seen in normal pressure hydrocephalus and bilateral chronic subdural hematoma [2].

High impact information on Hematoma, Subdural, Chronic

• Tissue plasminogen activator in chronic subdural hematomas as a predictor of recurrence [3].
• Computed tomography scan of the brain revealed a large heterogeneous subdural fluid collection over the left cerebral hemisphere, causing effacement of the adjacent sulci and a large left-to-right midline shift, consistent with an acute on chronic subdural hematoma [4].
• The authors investigated thrombomodulin expression to clarify why chronic subdural hematomas (CSDHs) continue to grow slowly, like a tumor, and are liquefied [5].
• On the other hand, patients who had chronic subdural hematomas with only slight headaches had the same phosphocreatine-to-inorganic phosphate ratio and brain intracellular pH as healthy volunteers [6].
• Plasmin-alpha 2-plasmin inhibitor complex and alpha 2-plasmin inhibitor in chronic subdural hematoma [7].

Chemical compound and disease context of Hematoma, Subdural, Chronic

• Coronal computerized angiotomography for the diagnosis of isodense chronic subdural hematoma [8].
• CONCLUSION: The phosphocreatine-to-inorganic phosphate ratio may be useful for determining when to operate on patients with chronic subdural hematomas and to assess the efficacy of treatment [6].
• Perioperative lumbar injection of Ringer's lactate solution in chronic subdural hematomas: a series of 100 cases [9].
• Rate constant of gadolinium (Gd)-DTPA transfer into chronic subdural hematomas [10].
• Steroid receptors in the pathogenesis of chronic subdural hematoma [11].

Gene context of Hematoma, Subdural, Chronic

• The present study was performed to investigate the relationship between the concentrations of tissue-type plasminogen activator (t-PA) and plasminogen activator inhibitor (PAI) and the CT images in 23 cases of chronic subdural hematomas (SDHs) [12].
• Relationship between tissue plasminogen activator, plasminogen activator inhibitor and CT image in chronic subdural hematoma [12].
• Increased concentration of vascular endothelial growth factor (VEGF) in chronic subdural hematoma [13].
• A computerized tomography scan revealed a left-sided chronic subdural hematoma and a midline shift to the right [14].
• Dural involvement in rheumatoid arthritis may occur in the absence of other rheumatoid lesions of the CNS; it appears to take a clinically uneventful course and does not seem to represent a factor predisposing to the development of chronic subdural hematoma [15].

References