Disease relevance of Pupil Disorders

• In patients with an intracranial hematoma and sudden onset of coma, unilateral fixed pupil was the most common initial manifestation [1].
• The complications of orbital cellulitis, occurring in five patients, included osteomyelitis of the maxillary bone, strabismus, afferent pupillary defect, chronic draining sinus, and scarred upper eyelid [2].
• Other ocular findings were exotropia, an ectopic pupil, band keratopathy, choroidal folds, anomalous optic discs and infantile myopia [3].
• The prediction of outcome of patients admitted following head injury in coma with bilateral fixed pupils [4].

High impact information on Pupil Disorders

• Coma, Glasgow Coma Score lower than 9, weakness score greater than 15 of a possible 30, abnormal ocular movements, and fixed pupils were also associated with stroke-related mortality [5].
• Dilated fixed pupils due to administration of high doses of dopamine hydrochloride [6].
• Dominantly inherited ptosis, strabismus and ectopic pupils [7].
• All patients at presentation, had full ophthalmological examination, fluorescein angiography, relative afferent pupillary defect test and electroretinography to distinguish between ischaemic and non-ischaemic occlusions [8].
• Only slight afferent pupillary defect was evident even 2 years after the onset of LHON [9].

Chemical compound and disease context of Pupil Disorders

• The pupillometer also exhibited reasonable accuracy in fixed pupils dilated with tropicamide [10].

Biological context of Pupil Disorders

• A healthy young white female presented with acute, unilateral loss of visual field and an afferent pupillary defect, but normal visual acuity, color vision, fundus examination, and fluorescein angiogram [11].

Gene context of Pupil Disorders

• The reaction was dominated by lens epithelial proliferation, pupillary occlusion, and glaucoma in 11 of the 20 [12].
• Marcus Gunn pupil in a possible case of unilateral retinitis pigmentosa [13].
• The CS at a natural pupil diameter and the MTF computed for a fixed pupil diameter were found to be significantly poorer for the older group than for the younger group [14].
• Four cases of pupillary paralysis are presented which followed implantation of the Surgidev version of the Choyce Mark VIII implant [15].
• Relative afferent pupillary defects were found in 69 of 386 patients (18%) seen by the author at the University of British Columbia Multiple Sclerosis Clinic between Feb. 1, 1985, and Apr. 30, 1987 [16].

References