Disease relevance of Acalculous Cholecystitis

• Among connective tissue disorders, acute acalculous cholecystitis has been reported in patients with polyarteritis nodosa and/or SLE [1].
• Systemic lupus erythematosus, eosinophilic vasculitis and acalculous cholecystitis [2].
• Diabetes, malignant disease, abdominal vasculitis, congestive heart failure, cholesterol embolization, and shock or cardiac arrest have been associated with acute acalculous cholecystitis [3].
• Acute acalculous cholecystitis associated with systemic lupus erythematosus with Sjogren's syndrome [4].
• We reviewed 57 patients, who during the last four years had cholecystokinin cholecystography during evaluation of abdominal pain, and found this test to be reliable for diagnosing chronic acalculous cholecystitis [5].

High impact information on Acalculous Cholecystitis

• Acute acalculous cholecystitis induced by lysophosphatidylcholine [6].
• This study investigated the use of a corn oil emulsion as an inexpensive alternative to sincalide in the scintigraphic diagnosis of chronic acalculous cholecystitis (CAC) [7].
• Cholecystokinin sonography: lack of utility in diagnosis of acute acalculous cholecystitis [8].
• Forty-one proved cases of acute acalculous cholecystitis imaged with technetium-99m iminodiacetic acid (IDA) cholescintigraphy were retrospectively analyzed [9].
• Acute acalculous cholecystitis: sensitivity in detection using technetium-99m iminodiacetic acid cholescintigraphy [9].

Chemical compound and disease context of Acalculous Cholecystitis

• Cholesterol stones were found in 74 patients, pigment stones in 21, and acalculous cholecystitis in 5 [10].
• Patients who receive long-term parenteral nutrition have an increased incidence of both calculous and acalculous cholecystitis [11].
• In experimental acalculous cholecystitis produced in dog, the levels of eicosanoids (except LTB4) were significantly changed [12].
• Studies on the etiology of acute acalculous cholecystitis: the effect of lipopolysaccharide on human gallbladder mucosal cells [13].
• Recent reports in the surgical literature state that persistent dense visualization of an otherwise normal-appearing gallbladder 36 hr after the administration of iopanoic acid is highly suggestive of acalculous cholecystitis [14].

Anatomical context of Acalculous Cholecystitis

• Lack of contraction of the gallbladder after injection of cholecystokinin should not be considered a major criterion in the diagnosis of acute acalculous cholecystitis [8].

Gene context of Acalculous Cholecystitis

• Acute acalculous cholecystitis with a decrease in CD4/CD8 ratio [15].
• The impaired gallbladder emptying in chronic acalculous cholecystitis appears to be due to diminished spontaneous contractile activity and decreased contractile responsiveness to both CCK and EFS [16].
• IDA cholescintigraphy is an important new modality for the diagnosis of acute acalculous cholecystitis which, in the past, has often been difficult to diagnose [17].
• Medication effects, benign postoperative jaundice, sepsis, TPN, and acalculous cholecystitis are responsible for intrahepatic cholestasis and conjugated hyperbilirubinemia [18].

Analytical, diagnostic and therapeutic context of Acalculous Cholecystitis

• Our findings suggest that an intravenous infusion of Moripron could be used for the prophylaxis of acute acalculous cholecystitis and sludge formation due to reduced biliary motility in patients on total parenteral nutrition [19].
• Cholecystokinin cholecystography, sonography, and scintigraphy: detection of chronic acalculous cholecystitis [20].
• Chronic acalculous cholecystitis: reproduction of pain with cholecystokinin and relief of symptoms with cholecystectomy [21].

References