Disease relevance of Tibial Arteries

• Thrombosis of tibial arteries in a patient receiving tamoxifen therapy [1].
• In 4 adult human amputated legs, diseased peroneal or tibial arteries were treated with a significant reduction in the percent luminal diameter stenosis from 62.75 prior to intervention to 36.5 following intervention (p = 0.021) [2].

High impact information on Tibial Arteries

• Adenosine and the adenine nucleotides caused a greater relaxation of strips of canine saphenous vein and tibial artery when they had been contracted by nerve stimulation than by exogenous norepinephrine [3].
• Gadolinium-enhanced magnitude contrast MR angiography of popliteal and tibial arteries [4].
• In the posterior tibial artery, the diameter increased significantly at the highest isoprenaline dose but changes in velocity and volume were not significant [5].
• The functional consequences were studied of exposing the dog tibial artery for 2 hr to 5-hydroxytryptamine (10(-6) M) in vitro [6].
• In so doing, the indoleamine inhibits adrenergic neurotransmission in the tibial artery by its action at prejunctional serotonergic receptors [6].

Biological context of Tibial Arteries

• Changes in 1) heart rate, 2) brachial blood pressure, and 3) ankle systolic blood pressure, have been measured together with 4) diameter, 5) mean blood velocity, and 6) mean volume flow in the common femoral and posterior tibial arteries in normal volunteers following intravenous infusions of isoprenaline [5].
• Lacidipine was 79-times more effective than mibefradil in reducing the vasoconstriction in isolated human anterior tibial artery [7].

Anatomical context of Tibial Arteries

• The authors compared the effect of furosemide and morphine on isolated rings of canine pulmonary artery (PA) and vein (PV) and mesenteric, splenic and anterior tibial arteries and their corresponding veins precontracted with norepinephrine or (15S)-hydroxy-11 alpha, 9 alpha-(epoxymethano)prosta-5Z,13E-dienoic acid [8].
• The effects of prostaglandin A2 (PGA2) and prostaglandin B2 (PGB2) on vascular smooth muscle tone, electrolyte movements and responses to vasoactive stimuli were evaluated with superfused canine tibial arteries [9].
• In isolated nutrient tibial arteries, endothelin-1 caused concentration-dependent contractions of rings with and without endothelium [10].

Associations of Tibial Arteries with chemical compounds

• The total sulfhydryl content of rabbit renal and canine tibial arteries and was not affected by norepinephrine or potassium chloride [11].
• These results suggest that 5-hydroxytryptamine is accumulated and released by tibial artery adrenergic nerves as a cotransmitter [6].
• In this study normalized L-T curves were obtained from rings of the dog anterior tibial artery at low (approximately ED50) and high (maximal activation) concentrations of potassium (K+), norepinephrine (NE), and calcium (Ca2+) [12].
• Longitudinal tensions were recorded from in situ isolated segments of dog anterior tibial arteries during vasoconstrictor responses [13].
• During the exposure, unmetabolized 5-hydroxytryptamine was accumulated by the cocaine-sensitive amine uptake mechanism of tibial artery adrenergic nerves [6].

Gene context of Tibial Arteries

• Endothelin receptor agonists and antagonists were used in two different models: isolated nutrient tibial arteries in organ bath and in vitro-perfused canine tibial bones [10].
• Occlusion of the popliteal and tibial artery complicating multiple exostoses [14].
• In May 1988, she had four large feeder vessels off the posterior tibial artery and two off the anterior tibial artery embolized by use of 1,000 microns PVA particles, occlusive spring coils, and detachable balloons [15].

References