High impact information on Lateral Thalamic Nuclei

• There were no significant alterations in nicotine binding in schizophrenia, and in DLB, a trend towards moderate reductions in most nuclei reached significance in the lateral dorsal nucleus [1].
• In the diencephalon, the magnocellular nucleus, the suprachiasmatic nucleus, the posterior tuberculum, and the ventrolateral and lateral thalamic nuclei were moderately to densely innervated with ghrelin-containing fibers [2].
• In contrast, in CA3 of the hippocampus and laterodorsal thalamic nucleus (LD), there were no gender differences in neuronal loss between these nTg groups [3].
• Visual responses in suprasylvian cortex were recorded before, during, and after the deactivation of the lateral part of the lateral posterior nucleus accomplished by the injection of lidocaine or GABA [4].
• From PND11 to PND16, thionin facilitates parcellation by extensive staining of dendritic processes of MGd, MGm, and lateral posterior nucleus neurons but not neurons of the MGv or the dorsal lateral geniculate nucleus [5].

Biological context of Lateral Thalamic Nuclei

• The striate cortical projections cover approximately the lateral two-thirds of the lateral posterior nucleus, overlapping a small retinal terminal field, and naso-temporal axes in the visual field are represented onto the cortico-recipient zone in a mirror-symmetric direction to that of the adjoining DLGN [6].

Anatomical context of Lateral Thalamic Nuclei

• On the other hand, unilateral lesions to the superficial layers of the superior colliculus produced a dramatic reduction in substance P immunoreactivity in the ipsilateral nucleus of the optic tract, lateral posterior nucleus, and dorsal and ventral lateral geniculate nuclei [7].
• The mediorostral lateral posterior nucleus, subparafascicular, lateral geniculate and habenular nuclei also contained calretinin messenger RNA probe label [8].
• Predominant PV immunostaining characterizes primary somatosensory, visual and auditory nuclei, the ventral lateral posterior nucleus, reticular nucleus (R), and to a lesser degree also, lateral part of the centre median nucleus, and anterior, lateral, and inferior divisions of the pulvinar complex [9].
• Layer V pyramidal cells in cortical areas 17 and 18a were found to project to the rostral portion of the lateral posterior nucleus (LTP) (the cortico-recipient zone) [10].
• Presence of a projection containing adenosine deaminase (ADA)-like immunoreactivity from the stratum opticum (SO) to the dorsomedial portion of the lateral posterior nucleus of the thalamus (LPN) of the rat was demonstrated using a method combining retrograde tracing by horseradish peroxidase (HRP) and immunohistochemistry for ADA [11].

Associations of Lateral Thalamic Nuclei with chemical compounds

• The laminar distribution of cortical connections with the tecto- and cortico-recipient zones in the cat's lateral posterior nucleus [12].
• These results suggest that transneuronal changes in the distribution of substance P in collicular neurons observed after enucleation could be reflected in their projections to the other primary visual centers and to the lateral posterior nucleus [7].
• The 5-HT innervation of the lateral nuclear group as well as that of the medial and lateral geniculate nuclei ranged from very weak to dense [13].
• RESULTS: Autoradiographic analysis revealed an increase in glucose utilization by several brain regions; the most consistent increase was found in the lateral posterior thalamic nucleus and pretectal region [14].
• To determine if labeled cells in the dense band were also projection neurons, WGA-HRP was injected into the lateral posterior nucleus and these sections were double-labeled with the glutamate antibody [15].

Gene context of Lateral Thalamic Nuclei

• The lateral dorsal nucleus of the bed nuclei of the stria terminalis (BST-LD) expresses dense oxytocin binding while lower binding is detected in the medial anterior BST (BST-MA) and adjacent ventrolateral septum (VLS) [16].
• Some epileptic rats showed increased Zif268 immunoreactivity in neurons of the ipsilateral ventral lateral and central lateral thalamic nuclei and increased Zif268 and Fos-like immunoreactivity in the pontine nuclei [17].
• The beta-actin mRNA level in ischemic controls was significantly increased in the dentate gyrus, habenular nucleus, and medial and lateral thalamic nuclei, where some afferent nerves project into the hippocampal pyramidal cells [18].
• These activation of PKC differentiates between [3H]MK-801 binding of HAS and LAS rats in frontal cortex (layer II-IV and cingulate), caudate-putamen, and ventral lateral thalamic nuclei [19].
• Injection of HRP into the lateral posterior nucleus labeled many neurons in the medial portion of the SO where medium-sized neurons with ADA immunoreactivity were concentrated [11].

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